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Toxic effects of expanded ataxin-1 involve mechanical instability of the nuclear membrane

  • Published:2012-06 Issue:6 Volume:1822 Page:906-917
  • ISSN:0925-4439
  • Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
  • language:en
  • Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Mapelli Lisa,Canale Claudio,Pesci Daniela,Averaimo Stefania,Guizzardi Fabiana,Fortunati Valentina,Falasca Laura,Piacentini Mauro,Gliozzi Alessandra,Relini Annalisa,Mazzanti Michele,Jodice Carla

Funder

Italian Ministero dell'Istruzione, Università e Ricerca

Universities of Genoa, Milan and Rome “Tor Vergata”

Italian Ministero della Salute (Ricerca corrente e finalizzata)

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference46 articles.

1. Pathogenic mechanisms of a polyglutamine-mediated neurodegenerative disease, spinocerebellar ataxia type 1;Zoghbi;J. Biol. Chem.,2009

2. Phenotypic effects of expanded ataxin-1 polyglutamines with interruptions in vitro;Calabresi;Brain Res. Bull.,2001

3. Evidence for a mechanism predisposing to intergenerational CAG repeat instability in spinocerebellar ataxia type I;Chung;Nat. Genet.,1993

4. Population variation analysis at nine loci containing expressed trinucleotide repeats;Jodice;Ann. Hum. Genet.,1997

5. Effect of trinucleotide repeat length and parental sex on phenotypic variation in spinocerebellar ataxia I;Jodice;Am. J. Hum. Genet.,1994

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