Mitochondrial permeability transition pore is a potential drug target for neurodegeneration-Reference-Cited by-同舟云学术

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Mitochondrial permeability transition pore is a potential drug target for neurodegeneration

Published:2014-08 Issue:8 Volume:1842 Page:1267-1272
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Rao Valasani Koteswara,Carlson Emily A.,Yan Shirley Shidu

Funder

National Institute of Aging

National Institute of General Medical Science

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference94 articles.

1. Early deficits in synaptic mitochondria in an Alzheimer's disease mouse model;Du;Proc. Natl. Acad. Sci. U. S. A.,2010

2. Synaptic mitochondrial pathology in Alzheimer's disease;Du;Antioxid. Redox Signal.,2011

3. Role of mitochondrial amyloid-beta in Alzheimer's disease;Chen;J. Alzheimers Dis.,2010

4. Mitochondrial Abeta: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease;Caspersen;FASEB J.,2005

5. Amyloid beta, mitochondrial dysfunction and synaptic damage: implications for cognitive decline in aging and Alzheimer's disease;Reddy;Trends Mol. Med.,2008

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