Activation of AKT signaling promotes cell growth and survival in α7β1 integrin-mediated alleviation of muscular dystrophy
Author:
Funder
NIH
Muscular Dystrophy Association
Ellison Medical Foundation
Publisher
Elsevier BV
Subject
Molecular Biology,Molecular Medicine
Reference55 articles.
1. Isolation of candidate cDNAs for portions of the Duchenne muscular dystrophy gene;Monaco;Nature,1986
2. The molecular basis of muscular dystrophy in the mdx mouse: a point mutation;Sicinski;Science,1989
3. Dystrophin–glycoprotein complex: its role in the molecular pathogenesis of muscular dystrophies;Matsumura;Muscle Nerve,1994
4. Three muscular dystrophies: loss of cytoskeleton–extracellular matrix linkage;Campbell;Cell,1995
5. A new model for the interaction of dystorphin with F-actin;Rybakova;J. Cell Biol.,1996
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