Increased pulmonary vascular expression of receptor for advanced glycation end products (RAGE) in experimental congenital diaphragmatic hernia
Author:
Publisher
Elsevier BV
Subject
General Medicine,Pediatrics, Perinatology and Child Health,Surgery
Reference24 articles.
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3. Airway smooth muscle changes in the nitrofen-induced congenital diaphragmatic hernia rat model;Belik;Pediatr Res,2003
4. Active collagen synthesis by pulmonary arteries in pulmonary hypertension complicated by congenital diaphragmatic hernia;Yamataka;J Pediatr Surg,1997
5. Soluble receptor for advanced glycation end products (sRAGE) attenuates haemodynamic changes to chronic hypoxia in the mouse;Farmer;Pulm Pharmacol Ther,2014
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1. Exploring new perspectives on congenital diaphragmatic hernia: A comprehensive review;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-04
2. The role of RAGE in host pathology and crosstalk between RAGE and TLR4 in innate immune signal transduction pathways;The FASEB Journal;2020-11
3. Expression of soluble receptor for advanced glycation end products is associated with disease severity in congenital diaphragmatic hernia;American Journal of Physiology-Lung Cellular and Molecular Physiology;2019-06-01
4. Pulmonary hypertension secondary to congenital diaphragmatic hernia: factors and pathways involved in pulmonary vascular remodeling;Pediatric Research;2019-02-19
5. Expression of Nitric Oxide Synthase Interacting Protein (NOSIP) is Decreased in the Pulmonary Vasculature of Nitrofen-Induced Congenital Diaphragmatic Hernia;European Journal of Pediatric Surgery;2018-12-10
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