Specific and redundant roles of PKBα/AKT1 and PKBβ/AKT2 in human pancreatic islets-Reference-Cited by-同舟云学术

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Specific and redundant roles of PKBα/AKT1 and PKBβ/AKT2 in human pancreatic islets

Published:2015-10 Issue:1 Volume:338 Page:82-88
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Dietrich Maren G.,Zuellig Richard A.,Spinas Giatgen A.,Lehmann Roger,Tschopp Oliver,Niessen Markus

Funder

Forschungskredit of the University of Zurich

Amélie Waring Foundation

Publisher

Elsevier BV

Subject

Cell Biology

Reference35 articles.

1. Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes;Butler;Diabetes,2003

2. Islet beta cell failure in type 2 diabetes;Prentki;J. Clin. Invest.,2006

3. Beta cell dysfunction and insulin resistance;Cerf;Front. Endocrinol.,2013

4. Pathophysiology and treatment of type 2 diabetes: perspectives on the past, present, and future;Kahn;Lancet,2014

5. On the role of IRS2 in the regulation of functional beta-cell mass;Niessen;Archiv. Physiol. Biochem.,2006

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Akt1 signalling supports acinar proliferation and limits acinar‐to‐ductal metaplasia formation upon induction of acute pancreatitis;The Journal of Pathology;2019-11-22

2. Replicative capacity of β-cells and type 1 diabetes;Journal of Autoimmunity;2016-07

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