The cyanobacterial neurotoxin β-N-methylamino-l-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells-Reference-Cited by-同舟云学术

The cyanobacterial neurotoxin β-N-methylamino-l-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells

Published:2019-06 Issue:2 Volume:379 Page:172-181
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Cheng Fang,Fransson Lars-Åke,Mani Katrin

Funder

Swedish Cancer Foundation

Demensförbundet

Vetenskapsrådet

Publisher

Elsevier BV

Subject

Cell Biology

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4. Environmental neurotoxin interaction with proteins: dose-dependent increase of free and protein-associated BMAA (β-N-methylamino-L-alanine) in neonatal rat brain;Karlsson;Sci. Rep.,2015

5. Dietary exposure to an environmental toxin triggers neurofibrillary tangles and amyloid deposits in the brain;Cox;Proc. R Soc. B,2016

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1. Interplay between glypican-1, amyloid-β and tau phosphorylation in human neural stem cells;Neuroscience;2024-08

2. Interplay between Glypican-1, Amyloid-B and Tau Phosphorylation in Human Neural Stem Cells;2024

3. Proteomic Investigation of Differential Interactomes of Glypican 1 and a Putative Disease-Modifying Variant of Ataxia;Journal of Proteome Research;2023-08-16

4. Complex modulation of cytokine-induced α-synuclein aggregation by glypican-1-derived heparan sulfate in neural cells;Glycobiology;2021-12-07

5. Isolation and Characterization of Heparan Containing Precursor Protein Degradation Products;Methods in Molecular Biology;2021-10-10