NUPR1 promotes the proliferation and migration of breast cancer cells by activating TFE3 transcription to induce autophagy-Reference-Cited by-同舟云学术

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NUPR1 promotes the proliferation and migration of breast cancer cells by activating TFE3 transcription to induce autophagy

Published:2022-09 Issue:1 Volume:418 Page:113234
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Xiao Heng,Long Jing,Chen Xiang,Tan Mi-Duo^ORCID

Publisher

Elsevier BV

Subject

Cell Biology

Reference31 articles.

1. Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries;Bray;CA A Cancer J. Clin.,2018

2. Health care services use among long-term breast cancer survivors: a systematic review;Jansana;J. Canc. Survivor. : Res. Pract.,2019

3. Breast cancer-derived exosomes regulate cell invasion and metastasis in breast cancer via miR-146a to activate cancer associated fibroblasts in tumor microenvironment;Yang;Exp. Cell Res.,2020

4. Autophagy-independent lysosomal targeting regulated by ULK1/2-FIP200 and ATG9;Goodwin;Cell Rep.,2017

5. Crosstalk between apoptosis, necrosis and autophagy;Nikoletopoulou;Biochim. Biophys. Acta,2013

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1. Mechanistic study of NUPR1 in bladder cancer development through transcriptional regulation of CCR2;Journal of Cellular Physiology;2024-08-16

2. Single-cell atlas profiling revealed cellular characteristics and dynamic changes after PD-1 blockade therapy of brain metastases from laryngeal squamous cell carcinoma;Molecular and Cellular Biochemistry;2024-08-01

3. NUPR1 induces autophagy and promotes the progression of Esophageal squamous cell carcinoma via the MAPK-mTOR pathway;Pathology - Research and Practice;2024-05

4. NUPR1 packaged in extracellular vesicles promotes murine triple-negative breast cancer in a type 1 interferon-independent manner;Extracellular Vesicles and Circulating Nucleic Acids;2024-01-16

5. NUPR1 contributes to activate TFE3-dependent autophagy leading to cervical cancer proliferation;Heliyon;2024-01

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