Sevoflurane preconditioning protects against acute MI/R injury via enhancing AdipoR1-Cav3 interaction and alleviating endoplasmic reticulum stress-Reference-Cited by-同舟云学术

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Sevoflurane preconditioning protects against acute MI/R injury via enhancing AdipoR1-Cav3 interaction and alleviating endoplasmic reticulum stress

Published:2022-08 Issue:1 Volume:417 Page:113217
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Zhang Yan-Qing^ORCID,Li Rong,Tian Shou-Yuan,Lv Jie-Ping,Yang Bao-Zhong,Wang Jin,Wang Li,Bai Xiao-Jie,Wang Chun-Hui,Wang Qiang

Publisher

Elsevier BV

Subject

Cell Biology

Reference16 articles.

1. Myocardial protection by isoflurane vs. sevoflurane in ultra-fast-track anaesthesia for off-pump aortocoronary bypass grafting;Hemmerling;Eur. J. Anaesthesiol.,2008

2. Preconditioning by sevoflurane decreases biochemical markers for myocardial and renal dysfunction in coronary artery bypass graft surgery: a double-blinded, placebo-controlled, multicenter study;Julier;Anesthesiology,2003

3. Sevoflurane preconditioning attenuates myocardial ischemia/reperfusion injury via caveolin-3-dependent cyclooxygenase-2 inhibition;Zhao;Circulation,2013

4. Essential role of caveolin-3 in adiponectin signalsome formation and adiponectin cardioprotection;Wang;Arterioscler. Thromb. Vasc. Biol.,2012

5. Hyperglycemia abrogates ischemic postconditioning cardioprotection by impairing AdipoR1/caveolin-3/STAT3 signaling in diabetic rats;Li;Diabetes,2016

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The pathological mechanisms and potential therapeutic drugs for myocardial ischemia reperfusion injury;Phytomedicine;2024-07

2. Sevoflurane alleviates inflammation, apoptosis and permeability damage of human umbilical vein endothelial cells induced by lipopolysaccharide by inhibiting endoplasmic reticulum stress via upregulating RORα;Prostaglandins & Other Lipid Mediators;2024-06

3. Sevoflurane alleviates oxygen-glucose deprivation/reoxygenation-induced damage in HT22 cells by activating the Keap1/Nrf2/ARE pathway to inhibit oxidative stress;International Journal of Neuroscience;2023-12-18

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