Desmoglein 3 – Influence on oral carcinoma cell migration and invasion-Reference-Cited by-同舟云学术

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Desmoglein 3 – Influence on oral carcinoma cell migration and invasion

Published:2018-09 Issue:2 Volume:370 Page:353-364
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Hoque Apu Ehsanul,Akram Saad Ullah,Rissanen Jouni,Wan Hong,Salo Tuula

Funder

Sigrid Juselius Foundation

Finnish Cancer Foundation

Medical Faculty of the University of Oulu

Oulu University Hospital

Medical Research Center Oulu

Finnish Doctoral Programme in Oral Sciences

Tyyni Tani Foundation

Oulu University Research Foundation

Centre for International Mobility

Publisher

Elsevier BV

Subject

Cell Biology

Reference62 articles.

1. Desmoglein 3: a help or a hindrance in cancer progression?;Brown;Cancers,2015

2. Nonjunctional human desmoglein 3 acts as an upstream regulator of Src in E-cadherin adhesion, a pathway possibly involved in the patho-genesis of pemphigus vulgaris;Tsang;J. Pathol.,2012

3. Desmoglein 3, via an interaction with E-cadherin, is associated with activation of Src;Tsang;PLoS One,2010

4. A molecular study of desmosomes identifies a desmoglein isoform switch in head and neck squamous cell carcinoma;Teh;J. Oral. Pathol. Med.,2011

5. Desmoglein 3 promotes cancer cell migration and invasion by regulating activator protein 1 and protein kinase C-dependent Ezrin activation;Brown;Oncogene,2014

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