cAMP effects in neuroendocrine tumors: The role of Epac and PKA in cell proliferation and adhesion-Reference-Cited by-同舟云学术

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cAMP effects in neuroendocrine tumors: The role of Epac and PKA in cell proliferation and adhesion

Published:2015-12 Issue:2 Volume:339 Page:241-251
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Vitali E.,Cambiaghi V.,Spada A.,Tresoldi A.,Zerbi A.,Peverelli E.,Carnaghi C.,Mantovani G.,Lania A.G.

Funder

Associazione Italiana Ricerca Cancro-Milan

Fondazione IRCCS Ca’ Granda Ospedale Maggiore Policlinico

ERA-NET “ERare”

Publisher

Elsevier BV

Subject

Cell Biology

Reference49 articles.

1. Functional roles of protein kinase A (PKA) and exchange protein directly activated by 3′,5′-cyclic adenosine 5′-monophosphate (cAMP) 2 (EPAC2) in cAMP-mediated actions in adrenocortical cells;Aumo;Endocrinology,2010

2. Differential effects of vasodilatory prostaglandins on focal adhesions, cytoskeletal architecture, and migration in human aortic smooth muscle cells;Bulin;Arteriosclerosis Thromb. Vasc. Biol.,2005

3. cAMP inhibits migration, ruffling and paxillin accumulation in focal adhesions of pancreatic ductal adenocarcinoma cells: effects of PKA and EPAC;Burdyga;Biochim. Et. Biophys. Acta,2013

4. cAMP antagonizes p21ras-directed activation of extracellular signal-regulated kinase 2 and phosphorylation of mSos nucleotide exchange factor;Burgering;EMBO J.,1993

5. The GTPase Rap1 controls functional activation of macrophage integrin alphaMbeta2 by LPS and other inflammatory mediators;Caron;Curr. Biol.,2000

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