MiR-106a-5p modulates apoptosis and metabonomics changes by TGF-β/Smad signaling pathway in cleft palate-Reference-Cited by-同舟云学术

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MiR-106a-5p modulates apoptosis and metabonomics changes by TGF-β/Smad signaling pathway in cleft palate

Published:2020-01 Issue:2 Volume:386 Page:111734
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Zhang Wancong,Shen Zhiwei,Xing Yue,Zhao Hanxing,Liang Yan,Chen Jiasheng,Zhong Xiaoping,Shi Lungang,Wan Xinhong,Zhou Jianda,Tang Shijie

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

The Scientific and Technology Innovation Project of Guangdong Province SHANFU

Science and Technology Project of Shantou

Publisher

Elsevier BV

Subject

Cell Biology

Reference33 articles.

1. Signaling networks in palate development;Lane;Wiley. Interdiscip. Rev. Syst. Biol. Med.,2014

2. Smad4-Irf6 genetic interaction and TGF -mediated IRF6 signaling cascade are crucial for palatal fusion in mice;Iwata;Development,2013

3. Palatogenesis: morphogenetic and molecular mechanisms of secondary palate development;Bush;Development,2012

4. Cleft lip and palate;Mossey;Lancet,2009

5. Genome-wide association study identifies a new susceptibility locus for cleft lip with or without a cleft palate;Sun;Nat. Commun.,2015

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1. Identification of RESP18 Gene Mutations Linked to Hereditary Non-Syndromic Cleft Lip and Palate in a Southern Chinese Family;Medical Science Monitor;2024-06-04

2. Differentially expressed microRNAs in aneuploid preimplantation blastocysts: a systematic review;Frontiers in Reproductive Health;2024-03-14

3. MicroRNAs in maxillofacial bone modeling and remodeling: implications for malocclusion development and orthodontic treatment;Frontiers in Cell and Developmental Biology;2024-03-13

4. Genetic variants in BCL‐2 family genes influence the risk of non‐syndromic cleft lip with or without cleft palate;Birth Defects Research;2023-12-18

5. Repurposing Niclosamide as a Therapeutic Drug against Acute Liver Failure by Suppressing Ferroptosis;Pharmaceutics;2023-07-14

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