MBD2-mediated Th17 differentiation in severe asthma is associated with impaired SOCS3 expression-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

MBD2-mediated Th17 differentiation in severe asthma is associated with impaired SOCS3 expression

Published:2018-10 Issue:1 Volume:371 Page:196-204
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Sun Wenjin,Xiao Bing,Jia Aijun,Qiu Lulu,Zeng Qingping,Liu Da,Yuan Yu,Jia Jingsi,Zhang Xiufeng,Xiang Xudong

Funder

National Natural Science Foundation of China

Scientific Research Project of Hunan Provincial Education Department

Publisher

Elsevier BV

Subject

Cell Biology

Reference35 articles.

1. Severe asthma: from characteristics to phenotypes to endotypes;Wenzel;Clin. Exp. Allergy.: J. Br. Soc. Allergy. Clin. Immunol.,2012

2. A summary of the new GINA strategy: a roadmap to asthma control;Reddel;Eur. Respir. J.,2015

3. Asthma phenotypes: the evolution from clinical to molecular approaches;Wenzel;Nat. Med.,2012

4. Wenzel SE.Pathobiology of severe asthma;Trejo Bittar;Annu. Rev. Pathol.,2015

5. Costs of asthma according to the degree of severity;Serra-Batlles;Eur. Respir. J.,1998

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Network pharmacology analysis and experimental validation of Xiao-Qing-Long-Tang’s therapeutic effects against neutrophilic asthma;Journal of Pharmaceutical and Biomedical Analysis;2024-06

2. Aberrant expression of suppressor of cytokine signaling (SOCS) molecules contributes to the development of allergic diseases;Clinical & Experimental Allergy;2023-08-28

3. miR-146a-3p as a potential novel therapeutic by targeting MBD2 to mediate Th17 differentiation in Th17 predominant neutrophilic severe asthma;Clinical and Experimental Medicine;2023-03-24

4. Methyl-cpg-binding Domain Protein 2 Silencing Inhibits Th17 Differentiation of CD4+T cells Induced by Ovalbumin;IRAN J IMMUNOL;2023

5. Methyl CpG binding domain protein 2 (MBD2) in inflammation;Chinese Medical Journal;2022-12-05

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3