High levels of endogenous nitric oxide produced after burn injury in rats arrest activated T lymphocytes in the first G1 phase of the cell cycle and then induce their apoptosis-Reference-Cited by-同舟云学术

High levels of endogenous nitric oxide produced after burn injury in rats arrest activated T lymphocytes in the first G1 phase of the cell cycle and then induce their apoptosis

Published:2005-05 Issue:1 Volume:306 Page:150-167
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Valenti Lionel M.,Mathieu Jacques,Chancerelle Yves,De Sousa Martine,Levacher Maryse,Tuan Dinh-Xuan Anh,Florentin Irène

Publisher

Elsevier BV

Subject

Cell Biology

Reference81 articles.

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4. Role of nitric oxide in depressed lymphoproliferative responses and altered cytokine production following thermal injury in rats;Masson;Cell. Immunol.,1998

5. Thermal injury alters macrophage responses to prostaglandin E2: contribution to the enhancement of inducible nitric oxide synthase activity;Schwacha;J. Leukocyte Biol.,1998

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