skNAC and Smyd1 in transcriptional control-Reference-Cited by-同舟云学术

skNAC and Smyd1 in transcriptional control

Published:2015-08 Issue:2 Volume:336 Page:182-191
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Berkholz Janine,Orgeur Mickael,Stricker Sigmar,Munz Barbara

Funder

Deutsche Forschungsgemeinschaft

German Foundation for Cardiac Research (DSHF)

German Research Foundation

Tubingen University Medical Clinic

Publisher

Elsevier BV

Subject

Cell Biology

Reference29 articles.

1. Skeletal muscle-specific variant of nascent polypeptide associated complex alpha (skNAC): implications for a specific role in mammalian myoblast differentiation;Berger;Eur. J. Cell Biol.,2012

2. skNAC depletion stimulates myoblast migration and perturbs sarcomerogenesis by enhancing calpain 1 and 3 activity;Berkholz;Biochem. J.,2013

3. The E3 SUMO ligase Nse2 regulates sumoylation and nuclear-to-cytoplasmic translocation of skNAC–Smyd1 in myogenesis;Berkholz;J. Cell Sci.,2014

4. Overexpression of the skNAC gene in human rhabdomyosarcoma cells enhances their differentiation potential and inhibits tumor cell growth and spreading;Berkholz;Clin. Exp. Metastasis,2014

5. Identification and characterization of Smyd2: a split SET/MYND domain-containing histone H3 lysine 36-specific methyltransferase that interacts with the Sin3 histone deacetylase complex;Brown;Mol. Cancer,2006

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