In Vitro Studies on the Effect of Cofactors on the 5α-Reductase and 3α- and 3β-hydroxysteroid Reductase Activities in the Hyperplastic Human Prostate
Author:
Affiliation:
1. From the Department of Surgery, Division of Urology, University of South Florida College of Medicine and Urology Section, Surgical Services, Bay Pines VAMC, Florida
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Urology
Reference17 articles.
1. Dihydrotestosterone in prostatic hypertrophy. I. The formation and content of dihydrotestosterone in the hypertrophic prostate of man;Siiteri;J. Clin. Invest.,1970
2. Comparison of androgen metabolites in benign prostatic hypertrophy (BPH) and normal prostate;Geller;J. Clin. Endocrinol. Metabol.,1976
3. Androgen levels in the plasma and prostatic tissue of patients with benign hypertrophy and carcinoma of the prostate;Habib;J. Endocrinol.,1976
4. Quantification of androgen binding, androgen tissue levels and sex-hormone binding globulin in prostate, muscle and plasma of patients with benign prostatic hypertrophy;Krieg;Acta Endocrinol. (Cophenh),1977
5. Endogenous steroid levels in the human prostate from birth to old age: a comparison of normal and diseased tissues;Hammond;J. Endocrinol.,1978
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1. Sequence of the cDNA of a human dihydrodiol dehydrogenase isoform (AKR1C2) and tissue distribution of its mRNA;Biochemical Journal;1998-09-01
2. Prevalent Decrease of the EGF Content in the Periurethral Zone of BPH Tissue Induced by Treatment With Finasteride or Flutamide;Journal of Andrology;1997-09-10
3. 3α-Hydroxysteroid oxidoreductase activities in dihydrotestosterone degradation and back-formation in rat prostate and epididymis;The Journal of Steroid Biochemistry and Molecular Biology;1996-06
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