GABA Synthesis and Metabolism
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Publisher
Elsevier
Reference26 articles.
1. Mice lacking the 65kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures;Asada;Biochemical and Biophysical Research Communications,1996
2. Cleft palate and decreased brain gamma-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase;Asada;Proceedings of the National Academy of Sciences of the United States of America,1997
3. The glutamate/GABA-glutamine cycle: Aspects of transport, neurotransmitter homeostasis and ammonia transfer;Bak;Journal of Neurochemistry,2006
4. Kinetic differences between the isoforms of glutamate decarboxylase: Implications for the regulation of GABA synthesis;Battaglioli;Journal of Neurochemistry,2003
5. Multiple G protein-coupled receptors initiate protein kinase C redistribution of GABA transporters in hippocampal neurons;Beckman;Journal of Neuroscience,1999
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