Prostate carcinoma (PC-3) cell proliferation is stimulated by the 22–25-kDa proteolytic fragment (1–160) and inhibited by the 16-kDa fragment (1–95) of recombinant human insulin-like growth factor binding protein-3
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Reference12 articles.
1. Insulin-like growth factors and their binding proteins: biological actions;Jones;Endocr Rev,1995
2. In vivo proteolysis of serum insulin-like growth factor (IGF) binding protein-3 results in increased availability of IGF to target cells;Blat;J Clin Invest,1994
3. Potentiation of insulin-like growth factor (IGF) action by IGF-binding protein-3: studies of underlying mechanism;Conover;Endocrinology,1992
4. Autocrine regulation of cell proliferation by the IGF and IGF binding protein-3 protease system in a human prostate carcinoma cell line (PC-3);Angelloz-Nicoud;Endocrinology,1995
5. IGF binding protein-3 secreted by prostate adenocarcinoma cells (PC-3): differential effect on PC-3 and normal prostate cell growth;Kaicer;Growth Regulation,1993
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