Long-term naringin consumption reverses a glucose uptake defect and improves cognitive deficits in a mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Long-term naringin consumption reverses a glucose uptake defect and improves cognitive deficits in a mouse model of Alzheimer's disease

Published:2012-07 Issue:1 Volume:102 Page:13-20
ISSN:0091-3057
Container-title:Pharmacology Biochemistry and Behavior
language:en
Short-container-title:Pharmacology Biochemistry and Behavior

Author:

Wang Dongmei,Gao Kai,Li Xiaoying,Shen Xinhua,Zhang Xu,Ma Chunmei,Qin Chuan,Zhang Lianfeng

Funder

National Science and Technology

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Biological Psychiatry,Clinical Biochemistry,Pharmacology,Toxicology,Biochemistry

Reference78 articles.

1. Inhibitory effect of naringin on the micronuclei induced by ifosfamide in mouse, and evaluation of its modulatory effect on the Cyp3a subfamily;Alvarez-Gonzalez;Mutat Res,2001

2. Alzheimer's disease and the basal forebrain cholinergic system: relations to beta-amyloid peptides, cognition, and treatment strategies;Auld;Prog Neurobiol,2002

3. Neuronal apoptosis in Alzheimer's disease: the role of age-related mitochondrial metabolic competence;Bertoni-Freddari;Ann N Y Acad Sci,2009

4. Naringenin has anti-inflammatory properties in macrophage and ex vivo human whole-blood models;Bodet;J Periodontal Res,2008

5. A GSK-3/TSC2/mTOR pathway regulates glucose uptake and GLUT1 glucose transporter expression;Buller;Am J Physiol Cell Physiol,2008

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