The α2C-adrenoceptor antagonist JP-1302 controls behavioral parameters, tyrosine hydroxylase activity and receptor expression in a rat model of ketamine-induced schizophrenia-like deficits

Author:

Tekin Nurdan,Karamahmutoğlu Tuğba Eryiğit,Aykaç Aslı,Akakın Dilek,Gören Mehmet Zafer

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Biological Psychiatry,Clinical Biochemistry,Pharmacology,Toxicology,Biochemistry

Reference62 articles.

1. Is the acute NMDA receptor hypofunction a valid model of schizophrenia?;Adell;Schizophr. Bull.,2012

2. The novel object recognition memory: neurobiology, test procedure, and its modifications;Antunes;Cogn. Process.,2012

3. GABAB/NMDA receptor interaction in the regulation of extracellular dopamine levels in rodent prefrontal cortex and striatum;Balla;Neuropharmacology,2009

4. Ketamine-induced changes in rat behaviour: a possible animal model of schizophrenia. Test of predictive validity;Becker;Prog. Neuro-Psychopharmacol. Biol. Psychiatry,2004

5. Ketamine-induced changes in rat behaviour: a possible animal model of schizophrenia;Becker;Prog. Neuro-Psychopharmacol. Biol. Psychiatry,2003

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