p38 MAP kinase as a therapeutic target in cardiovascular disease
Author:
Publisher
Elsevier BV
Subject
Drug Discovery,Pharmacology,Molecular Medicine
Reference21 articles.
1. Activation of the p38 MAP kinase pathway is required for foam cell formation from macrophages exposed to oxidized LDL;Zhao;Acta Pathol. Microbiol. Scand.,2002
2. OxLDL upregulates CXCR2 expression in monocytes via scavenger receptors and activation of p38 mitogen-activated protein kinase;Lei;Cardiovasc. Res.,2002
3. Native LDL induces interleukin-8 expression via H2O2, p38 Kinase, and activator protein-1 in human aortic smooth muscle cells;Ryoo;Cardiovasc. Res.,2004
4. Reactive oxygen species-sensitive p38 MAPK controls thrombin-induced migration of vascular smooth muscle cells;Wang;J. Mol. Cell. Cardiol.,2004
5. An inhibitor of p38 mitogen-activated protein kinase protects neonatal cardiac myocytes from ischemia;MacKay;J. Biol. Chem.,1999
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Protein kinases as cardiovascular therapeutic targets;The Lancet;2014-09
2. Inhibition of p38 MAPK During Ischemia, But Not Reperfusion, Effectively Attenuates Fatal Arrhythmia in Ischemia/Reperfusion Heart;Journal of Cardiovascular Pharmacology;2013-02
3. Age-induced augmentation of p38 MAPK phosphorylation in mouse lung;Experimental Gerontology;2011-05
4. Activation of mitogen activated protein kinases in post-infarcted patients;Journal of Thrombosis and Thrombolysis;2010-11-06
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