Mixed chimerism and transplant tolerance are not effectively induced in C3a-deficient mice-Reference-Cited by-同舟云学术

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Mixed chimerism and transplant tolerance are not effectively induced in C3a-deficient mice

Published:2015-01 Issue:1 Volume:43 Page:14-22
ISSN:0301-472X
Container-title:Experimental Hematology
language:en
Short-container-title:Experimental Hematology

Author:

Baśkiewicz-Hałasa Magdalena,Rogińska Dorota,Piecyk Katarzyna,Hałasa Maciej,Lejkowska Renata,Pius-Sadowska Ewa,Machaliński Bogusław

Funder

State Committee for Scientific Research

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Genetics,Hematology,Molecular Biology

Reference35 articles.

1. Complement genetics, deficiencies, and disease associations;Mayilyan;Protein Cell,2012

2. Role of complement and perspectives for interventions in transplantation;Chen;Immunobiology,2013

3. Transplantation studies in C3-deficient animals reveal a novel role of the third complement component (C3) in engraftment of bone marrow cells;Ratajczak;Leukemia,2004

4. Complement C3a enhances CXCL12 (SDF-1)-mediated chemotaxis of bone marrow hematopoietic cells independently of C3a receptor;Honczarenko;J Immunol,2005

5. Immuno-intervention for the induction of transplantation tolerance through mixed chimerism;Sachs;Semin Immunol,2011

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. C5aR1 regulates migration of suppressive myeloid cells required for costimulatory blockade‐induced murine allograft survival;American Journal of Transplantation;2018-09-17

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