Molecular Basis of Hematological Disease Caused by Inherited or Acquired RUNX1 Mutations
Author:
Funder
Bloodwise
Wellcome Trust
Blood Cancer UK
Leukaemia UK
Medical Research Council
Publisher
Elsevier BV
Subject
Cancer Research,Cell Biology,Genetics,Hematology,Molecular Biology
Reference127 articles.
1. The leukemia-associated AML1 (Runx1)—CBF beta complex functions as a DNA-induced molecular clamp;Bravo;Nat Struct Biol,2001
2. DNA Recognition by the RUNX1 transcription factor is mediated by an allosteric transition in the RUNT domain and by DNA bending;Bartfeld;Structure,2002
3. RUNX: a trilogy of cancer genes;Lund;Cancer Cell,2002
4. The RUNX genes: gain or loss of function in cancer;Blyth;Nat Rev Cancer,2005
5. RUNX1 cooperates with FLT3-ITD to induce leukemia;Behrens;J Exp Med,2017
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1. Diagnosing familial platelet disorder with predisposition to myeloid malignancy: Lessons learned from a germline whole‐gene deletion of RUNX1;International Journal of Laboratory Hematology;2023-11-12
2. Germline and somatic drivers in inherited hematologic malignancies;Frontiers in Oncology;2023-10-13
3. Interface-guided phenotyping of coding variants in the transcription factor RUNX1 with SEUSS;2023-08-04
4. Mechanisms of Secondary Leukemia Development Caused by Treatment with DNA Topoisomerase Inhibitors;Biochemistry (Moscow);2023-07
5. An Old Dog Has a New Trick: Somatic Exonic Deletions inRUNX1Are Frequent in AML;Clinical Cancer Research;2023-05-30
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