Key role of glutamine metabolism in persistence of leukemic cells upon exposition to FLT3 tyrosine kinase inhibitors-Reference-Cited by-同舟云学术

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Key role of glutamine metabolism in persistence of leukemic cells upon exposition to FLT3 tyrosine kinase inhibitors

Published:2024-09 Issue: Volume:137 Page:104253
ISSN:0301-472X
Container-title:Experimental Hematology
language:en
Short-container-title:Experimental Hematology

Author:

Khamari Raeeka,Degand Claire,Fovez Quentin^ORCID,Trinh Anne,Chomy Axel^ORCID,Laine William^ORCID,Dekiouk Salim^ORCID,Ghesquiere Bart,Quesnel Bruno^ORCID,Marchetti Philippe,Manier Salomon,Kluza J.^ORCID

Funder

Daiichi Sankyo Europe

Ligue Nationale Contre Le Cancer

Agence Régionale de Santé Hauts-de-France

Université de Lille

Inserm Transfert

Publisher

Elsevier BV

Reference36 articles.

1. Targeting FLT3 mutations in AML: review of current knowledge and evidence;Daver;Leukemia,2019

2. Validation of ITD mutations in FLT3 as a therapeutic target in human acute myeloid leukaemia;Smith;Nature,2012

3. Overcoming Resistance: FLT3 Inhibitors Past, Present, Future and the Challenge of Cure;Capelli;Cancers,2022

4. Tandem-duplicated Flt3 constitutively activates STAT5 and MAP kinase and introduces autonomous cell growth in IL-3-dependent cell lines;Hayakawa;Oncogene,2000

5. FLT3 and FLT3-ITD phosphorylate and inactivate the cyclin-dependent kinase inhibitor p27Kip1 in acute myeloid leukemia;Peschel;Haematologica,2017

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Glutamine and leukemia research: progress and clinical prospects;Discover Oncology;2024-08-31

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