Preclinical rationale for TGF-β inhibition as a therapeutic target for the treatment of myelofibrosis-Reference-Cited by-同舟云学术

Preclinical rationale for TGF-β inhibition as a therapeutic target for the treatment of myelofibrosis

Published:2016-12 Issue:12 Volume:44 Page:1138-1155.e4
ISSN:0301-472X
Container-title:Experimental Hematology
language:en
Short-container-title:Experimental Hematology

Author:

Ceglia Ilaria,Dueck Amylou C.,Masiello Francesca,Martelli Fabrizio,He Wu,Federici Giulia,Petricoin Emanuel F.,Zeuner Ann,Iancu-Rubin Camelia,Weinberg Rona,Hoffman Ronald,Mascarenhas John,Migliaccio Anna Rita

Funder

National Cancer Institute

Associazione Italiana Ricerca sul Cancro

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Genetics,Hematology,Molecular Biology

Reference46 articles.

1. Pathologic interaction between megakaryocytes and polymorphonuclear leukocytes in myelofibrosis;Schmitt;Blood,2000

2. Pivotal contributions of megakaryocytes to the biology of idiopathic myelofibrosis;Ciurea;Blood,2007

3. Evaluation of the bioactive and total transforming growth factor β1 levels in primary myelofibrosis;Campanelli;Cytokine,2011

4. Characterization of the TGF-beta1 signaling abnormalities in the Gata1low mouse model of myelofibrosis;Zingariello;Blood,2013

5. Prominent role of TGF-beta 1 in thrombopoietin-induced myelofibrosis in mice;Chagraoui;Blood,2002

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