Two independent amsacrine-resistant human myeloid leukemia cell lines share an identical point mutation in the 170 kDa form of human topoisomerase II

Author:

Lee Ming-Sheng,Wang James C.,Beran Miloslav

Publisher

Elsevier BV

Subject

Molecular Biology,Structural Biology

Reference33 articles.

1. DNA sequence of the E. coli gyrB gene: application of a new sequence strategy;Adachi;Nucl. Acids Res,1987

2. The production of topoisomerase II-mediated DNA cleavage in human leukemia cells predicts their susceptibility to 4′-(9-acridinylamino)methanesulfon-m-anisidide (m-AMSA);Bakic;Biochem. Biophys. Res. Common,1986

3. Development and characterization of a human myelogenous leukemia cell line resistant to 4′-(9-acridinylamino)-3-methanesulfon-m-anisidide;Beran;Cancer Res,1987

4. Non-intercalative antitumor drugs interfere with the breakage-reunion reaction of mammalian DNA topoisomerase II;Chen;Biol. Chem,1984

5. DNA binding by epipodophyllotoxins and n-acyl anthracyclines: implications for mechanism of topoisomerase II inhibition;Chow;Mol. Pharmacol,1988

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