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Signaling cell death from the endoplasmic reticulum stress response

Published:2011-04 Issue:2 Volume:23 Page:143-149
ISSN:0955-0674
Container-title:Current Opinion in Cell Biology
language:en
Short-container-title:Current Opinion in Cell Biology

Author:

Shore Gordon C,Papa Feroz R,Oakes Scott A

Funder

Canadian Institutes of Health Research

National Cancer Institute of Canada

National Institutes of Health: Director's New Innovator Award

Sandler Program in Basic Sciences

Burroughs Wellcome Foundation

Juvenile Diabetes Research Foundation

Partnership for Cures

HHMI Physician-Scientist Early Career Award

V Foundation

Steward Trust Foundation

Publisher

Elsevier BV

Subject

Cell Biology

Reference58 articles.

1. Protein quality control in the early secretory pathway;Anelli;EMBO J,2008

2. Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes;Oyadomari;J Clin Invest,2002

3. Coxsackievirus B3 infection activates the unfolded protein response and induces apoptosis through downregulation of p58IPK and activation of CHOP and SREBP1;Zhang;J Virol,2010

4. ER chaperone functions during normal and stress conditions;Ma;J Chem Neuroanat,2004

5. Signal integration in the endoplasmic reticulum unfolded protein response;Ron;Nat Rev Mol Cell Biol,2007

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