Histopathological and enzyme histochemical changes in experimental Semliki forest virus infection in mice and their relevance to scrapie
Author:
Publisher
Elsevier BV
Subject
General Veterinary,Pathology and Forensic Medicine
Reference21 articles.
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2. Mouse encephalitis induced by avirulent Semliki forest virus;Chew-Lim;Veterinary Pathology,1975
3. Demyelination in mice after two or three infections with avirulent Semliki forest virus;Chew-Lim;Veterinary Pathology,1977
4. The pathology of natural and experimental scrapie;Fraxer,1976
5. Histochemical demonstration of N-acetyl-β-d-glucosaminidase employing naphthol AS-BI N-acetyl-β-glucosaminide as substrate;Hayashi;Journal of Histochemistry and Cytochemistry,1965
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1. In Semliki Forest virus encephalitis, suppressor of cytokine signaling 4 (SOCS4) is an essential modulator of immune responses that mediates the balance between immunopathology and virus clearance;Immunology & Cell Biology;2023-02-14
2. Viral mouse models used to study multiple sclerosis: past and present;Archives of Virology;2021-02-13
3. Mouse models of alphavirus-induced inflammatory disease;Journal of General Virology;2015-02-01
4. Role of interferon-γ and nitric oxide in the neuropathogenesis of avirulent Semliki Forest virus infection;Neuropathology and Applied Neurobiology;2003-10-17
5. Characterization of the cellular and cytokine response in the central nervous system following Semliki Forest virus infection;Journal of Neuroimmunology;1997-04
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