Revisiting the mechanisms of copper toxicity to rainbow trout: Time course, influence of calcium, unidirectional Na+ fluxes, and branchial Na+, K+ ATPase and V-type H+ ATPase activities-Reference-Cited by-同舟云学术

Revisiting the mechanisms of copper toxicity to rainbow trout: Time course, influence of calcium, unidirectional Na+ fluxes, and branchial Na+, K+ ATPase and V-type H+ ATPase activities

Published:2016-08 Issue: Volume:177 Page:51-62
ISSN:0166-445X
Container-title:Aquatic Toxicology
language:en
Short-container-title:Aquatic Toxicology

Author:

Chowdhury M. Jasim,Girgis Mina,Wood Chris M.

Funder

Natural Sciences and Engineering Research Council (NSERC) of Canada

International Copper Association (ICA)

Copper Development Association (CDA)

International Lead Zinc Research Organization

Nickel Producers Environmental Research Association (NiPERA)

Noranda-Falconbridge (now Glencore)

Teck-Cominco (now Teck Resources)

Inco (now Vale)

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Aquatic Science

Reference75 articles.

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2. Copper intoxication in Chinook salmon (Oncorhynchus tshawytscha) induced by natural springwater: effects on gill Na+ K+-ATPase, hematocrit, and plasma glucose;Beckman;Can. J. Fish. Aquat. Sci.,1988

3. Sodium and chloride transport in soft water and hard water acclimated zebrafish (Danio rerio);Boisen;Biochim. Biophys. Acta,2003

4. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding;Bradford;Anal. Biochem.,1976

5. The effects of chloride, calcium, and dissolved organic carbon on silver toxicity: comparison between rainbow trout and fat-head minnows;Bury;Environ. Toxicol. Chem.,1999

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