Polycyclic aromatic hydrocarbon and hypoxia exposures result in mitochondrial dysfunction in zebrafish-Reference-Cited by-同舟云学术

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Polycyclic aromatic hydrocarbon and hypoxia exposures result in mitochondrial dysfunction in zebrafish

Published:2019-11 Issue: Volume:216 Page:105298
ISSN:0166-445X
Container-title:Aquatic Toxicology
language:en
Short-container-title:Aquatic Toxicology

Author:

Lindberg Casey D.,Di Giulio Richard T.

Funder

Duke University Superfund Research Center

Duke University Integrated Toxicology and Environmental Health Program

Water Resources Research Institute and North Carolina Sea Grant

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Aquatic Science

Reference64 articles.

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3. Mitochondria-targeted heme oxygenase-1 induces oxidative stress and mitochondrial dysfunction in macrophages, kidney fibroblasts and in chronic alcohol hepatotoxicity;Bansal;Redox Biol.,2014

4. Evaluating biological significance of chemical exposure to fish using a bioenergetics-based stressor-response model;Beyers;Can. J. Fish. Aquat. Sci.,2011

5. The developmental epigenomics toolbox: ChIP-seq and MethylCap-seq profiling of early zebrafish embryos;Bogdanović;Methods,2013

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3. Avermectin induced vascular damage in zebrafish larvae: association with mitochondria-mediated apoptosis and VEGF/Notch signaling pathway;Journal of Hazardous Materials;2024-09

4. Water-accommodated fractions of crude oil and its mixture with chemical dispersant impairs oxidase stress and energy metabolism disorders in Oryzias melastigma embryos;Chemosphere;2024-09

5. Co-exposure effects of urinary polycyclic aromatic hydrocarbons and metals on lung function: mediating role of systematic inflammation;BMC Pulmonary Medicine;2024-08-11

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