Effects of GM1 ganglioside upon neuronal degeneration during withdrawal from alcohol

Author:

Cadete-Leite A.,Brandāo F.,Madeira M.D.,Paula-Barbosa M.M.

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Neurology,Toxicology,General Medicine,Biochemistry,Health (social science)

Reference62 articles.

1. Long-term alcohol consumption reduces the number of neuronal nuclear pores;Andrade;Alcohol.: Clin. Exp. Res.,1988

2. Disposition of exogenous tritium-labelled GM1 monosialoganglioside in the rat;Bellato;Clin. Tri. J.,1989

3. HGM ganglioside enhances synaptosomal resistance to chemically induced damage;Bondy;Neurosci. Lett.,1988

4. Chronic alcohol consumption induces lipofuscin deposition in the rat hippocampus;Borges;Neurobiol. Aging,1986

5. The impact of recent stereological advances on quantitative studies of the nervous system;Braendgaard;J. Neurosci. Methods,1986

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