AAV-BDNF gene therapy ameliorates a hypothalamic neuroinflammatory signature in the Magel2-null model of Prader-Willi syndrome-Reference-Cited by-同舟云学术

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AAV-BDNF gene therapy ameliorates a hypothalamic neuroinflammatory signature in the Magel2-null model of Prader-Willi syndrome

Published:2023-12 Issue: Volume:31 Page:101108
ISSN:2329-0501
Container-title:Molecular Therapy - Methods & Clinical Development
language:en
Short-container-title:Molecular Therapy - Methods & Clinical Development

Author:

Queen Nicholas J.,Huang Wei,Zou Xunchang,Mo Xiaokui,Cao Lei^ORCID

Publisher

Elsevier BV

Subject

Genetics,Molecular Biology,Molecular Medicine

Reference74 articles.

1. Recommendations for the investigation of animal models of Prader-Willi syndrome;Resnick;Mamm. Genome,2013

2. Prader-willi syndrome: clinical aspects;Elena;J. Obes.,2012

3. Prader-Willi syndrome and cycloid psychoses;Verhoeven;J. Intellect. Disabil. Res.,1998

4. Prader–willi Syndrome: Atypical Psychoses and Motor Dysfunctions;Verhoeven,2006

5. A profile of mental health and behaviour in Prader-Willi syndrome;Feighan;J. Intellect. Disabil. Res.,2020

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MAGEL2 (patho‐)physiology and Schaaf–Yang syndrome;Developmental Medicine & Child Neurology;2024-07

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