High Levels of Frataxin Overexpression Lead to Mitochondrial and Cardiac Toxicity in Mouse Models-Reference-Cited by-同舟云学术

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High Levels of Frataxin Overexpression Lead to Mitochondrial and Cardiac Toxicity in Mouse Models

Published:2020-12 Issue: Volume:19 Page:120-138
ISSN:2329-0501
Container-title:Molecular Therapy - Methods & Clinical Development
language:en
Short-container-title:Molecular Therapy - Methods & Clinical Development

Author:

Belbellaa Brahim,Reutenauer Laurence,Messaddeq Nadia,Monassier Laurent,Puccio Hélène

Funder

Lefoulond Delalande Foundation–Institut de France

Friedreich Ataxia Research Alliance

French Muscular Dystrophy Association AFM-Téléthon

AAVLIFE SAS

Agence Nationale de la Recherche

Publisher

Elsevier BV

Subject

Genetics,Molecular Biology,Molecular Medicine

Reference76 articles.

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3. Friedreich’s ataxia: autosomal recessive disease caused by an intronic GAA triplet repeat expansion;Campuzano;Science,1996

4. DNA triplet repeats mediate heterochromatin-protein-1-sensitive variegated gene silencing;Saveliev;Nature,2003

5. Mammalian iron-sulfur cluster biogenesis: Recent insights into the roles of frataxin, acyl carrier protein and ATPase-mediated transfer to recipient proteins;Maio;Curr. Opin. Chem. Biol.,2020

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