Early postnatal cocaine exposure causes sequential, dose-dependent, enduring but reversible supersensitivity in 5-HT2A receptor-mediated function during development in male mice

Author:

Darmani Nissar A,Ahmad Bashir

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Developmental Neuroscience,Toxicology

Reference65 articles.

1. Prenatal cocaine exposure disrupts the development of the serotonergic system;Akbari;Brain Res,1992

2. Potentiation of 5-HT1A receptor-mediated neuroendocrine responses in male but not female rat progeny after prenatal cocaine;Battaglia;J Pharmacol Exp Ther,1994

3. Prenatal cocaine produces biochemical and functional changes in brain serotonin systems in rat progeny;Battaglia;NIDA Res Mongr Series,1995

4. Evidence for alterations in presynaptic serotonergic function during withdrawal from chronic cocaine in rats;Baumann;Eur J Pharmacol,1995

5. Withdrawal from chronic cocaine enhances behavioral sensitivity to the 5HT2/1C agonist DOI;Baumann;Biol Psychiatry,1993

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