Conversion From Cyclosporine to Tacrolimus for Chronic Allograft Nephropathy
Author:
Publisher
Elsevier BV
Subject
Transplantation,Surgery
Reference19 articles.
1. The pathobiology of chronic allograft nephropathy: immune-mediated damage and accelerated aging;Joosten;Kidney Int,2004
2. Advances in the study of chronic allograft rejection;Orosz;Current Opinion in Organ Transplantation,2003
3. Early prognosis of the development of renal chronic allograft rejection by gene expression profiling of human protocol biopsies;Scherer;Transplantation,2003
4. Cyclosporine induced renal dysfunction;Paul;Transplant Proc,2004
5. Clinical study on the course delay of grafted renal function by tacrolimus;Shu-ming;Chin J Organ Transplant,2000
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1. Effects of sequential application of immunosuppressive agents according to the cell cycle in adriamycin-induced nephropathy rats;EUR REV MED PHARMACO;2019
2. Tacrolimus in preventing transplant rejection in Chinese patients – optimizing use;Drug Design, Development and Therapy;2015-01
3. FK506 inhibits the mice glomerular mesangial cells proliferation by affecting the transforming growth factor-βand Smads signal pathways;Renal Failure;2014-02-10
4. Tacrolimus abrogates TGF-β1-induced type I collagen production in normal human fibroblasts through suppressing p38MAPK signalling pathway: implications on treatment of chronic atopic dermatitis lesions;Journal of the European Academy of Dermatology and Venereology;2013-01-10
5. FK506 inhibits the enhancing effects of transforming growth factor (TGF)-β1 on collagen expression and TGF-β/Smad signalling in keloid fibroblasts: implication for new therapeutic approach;British Journal of Dermatology;2012-07-19
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