Molecular mechanisms for the regulation of aryl sulfotransferase IV expression during 2-acetylaminofluorene-induced hepatocarcinogenesis in rat

Author:

Ringer David P.,Yerokun Tokunbo,Khan Akbar S.

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference28 articles.

1. Regression and persistence of hyperplastic nodules induced by N-2-fluorenylacetamide and their relationship to hepatocarcinogenesis;Teebor;Cancer Res.,1971

2. Changes in rat liver N-hydroxy-2-acetylaminofluorene aryl sulfotransferase activity at early and late stages of hepatocarcinogenesis resulting from dietary administration of 2-acetylaminofluorene;Ringer;Cancer Lett.,1988

3. 2-Acetylaminofluorene-mediated alteration in the level of liver aryl sulfotransferase IV during rat hepatocarcinogenesis;Ringer;Cancer Res.,1990

4. Changes in levels of ADP-ribose polymers in rat liver during 2-acetylaminofluorene-induced hepatocarcinogenesis;Kiehlbauch;Carcinogenesis,1993

5. Enzyme-catalyzed reactions of the carcinogen N-hydroxy-2-fluorenylacetamide with nucleic acid;King;Science (Washington DC),1968

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