AGEs induce endothelial cells senescence and endothelial barrier dysfunction via miR-1-3p/MLCK signaling pathways-Reference-Cited by-同舟云学术

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AGEs induce endothelial cells senescence and endothelial barrier dysfunction via miR-1-3p/MLCK signaling pathways

Published:2023-01 Issue: Volume:851 Page:147030
ISSN:0378-1119
Container-title:Gene
language:en
Short-container-title:Gene

Author:

Cheng Mian,Yang Zhen,Qiao Lifen,Yang Yi,Deng Yulei,Zhang Cuntai,Mi Tao

Funder

National Natural Science Foundation of China

National Key Research and Development Program of China

Publisher

Elsevier BV

Subject

Genetics,General Medicine

Reference48 articles.

1. Plasma carboxymethyl-lysine, an advanced glycation end product, and all-cause and cardiovascular disease mortality in older community-dwelling adults;Semba;J. Am. Geriatr. Soc,2009

2. Advanced glycation end products: sparking the development of diabetic vascular injury;Goldin;Circulation,2006

3. Does accumulation of advanced glycation end products contribute to the aging phenotype?;Semba;J. Gerontol. A. Biol. Sci. Med. Sci,2010

4. Relationships between advanced glycation end products (AGEs), vasoactive substances, and vascular function;Matsumoto;J. Smooth. Muscle. Res,2021

5. Endothelial aging;Brandes;Cardiovasc. Res,2005

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