GNAS gene is an important regulator of insulin secretory capacity in pancreatic β-cells

Author:

Taneera JalalORCID,Dhaiban Sarah,Mohammed Abdul KhaderORCID,Mukhopadhyay Debasmita,Aljaibeji Hayat,Sulaiman Nabil,Fadista Joao,Salehi AlbertORCID

Funder

University of Sharjah

Aljalila Foundation

Mats Paulsson foundation

Forget Foundation

Swedish Diabetes Foundation

Linnaeus

Strategic Research Area Exodiab

Publisher

Elsevier BV

Subject

Genetics,General Medicine

Reference59 articles.

1. The functional impact of G protein-coupled receptor 142 (Gpr142) on pancreatic β-cell in rodent;Al-Amily;Pflugers Arch. - Eur. J. Physiol.,2019

2. Novel observations from next generation RNA sequencing of highly purified human adult and fetal islet cell subsets;Blodgett;Diabetes,2015

3. Epigenomic plasticity enables human pancreatic α to β cell reprogramming;Bramswig;J. Clin. Invest.,2013

4. Pseudohypoparathyroidism: defective excretion of 3',5'-AMP in response to parathyroid hormone;Chase;J Clin Invest,1969

5. Increased insulin sensitivity in paternal Gnas knockout mice is associated with increased lipid clearance;Chen;Endocrinology,2004

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