Close homolog of L1-deficient ameliorates inflammatory bowel disease by regulating the balance of Th17/Treg-Reference-Cited by-同舟云学术

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Close homolog of L1-deficient ameliorates inflammatory bowel disease by regulating the balance of Th17/Treg

Published:2020-10 Issue: Volume:757 Page:144931
ISSN:0378-1119
Container-title:Gene
language:en
Short-container-title:Gene

Author:

Yao Jun,Gao Ruoyu,Luo Minghan,Li Defeng,Guo Liliangzi,Yu Zichao,Xiong Feng,Wei Cheng,Wu Benhua,Xu Zhenglei,Zhang Dingguo,Wang Jianyao,Wang Lisheng

Funder

National Natural Science Foundation of China

Three engineering training funds in Shenzhen

Technical Research and Development Project of Shenzhen

Publisher

Elsevier BV

Subject

Genetics,General Medicine

Reference41 articles.

1. The HDAC inhibitor, SAHA, prevents colonic inflammation by suppressing pro-inflammatory cytokines and Chemokines in DSS-induced Colitis;Ali;Acta Histochem Cytochem.,2018

2. Inflammatory bowel disease: clinical aspects and established and evolving therapies;Baumgart;Lancet,2007

3. The immunological and genetic basis of inflammatory bowel disease;Bouma;Nat. Rev. Immunol.,2003

4. Integrins and adhesion molecules as targets to treat inflammatory bowel disease;Bravatà;Curr. Opin. Pharmacol.,2015

5. Adhesion molecules in inflammatory bowel disease: therapeutic implications for gut inflammation;Danese;Dig. Liver Dis.,2005

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3. Shaoyao decoction alleviates TNBS-induced ulcerative colitis by decreasing inflammation and balancing the homeostasis of Th17/Treg cells;BMC Complementary Medicine and Therapies;2023-11-24

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