A novel activation process of protein kinase C in the remote, non-ischemic area of an infarcted heart is mediated by angiotensin-AT1 receptors
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine,Molecular Biology
Cited by 14 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Functional and prognostic implications of cardiac magnetic resonance feature tracking-derived remote myocardial strain analyses in patients following acute myocardial infarction;Clinical Research in Cardiology;2020-10-20
2. ACE Inhibition Prevents Diastolic Ca2+ Overload and Loss of Myofilament Ca2+ Sensitivity after Myocardial Infarction;Current Molecular Medicine;2012-02-01
3. Heterogeneity in MT1-MMP activity with ischemia-reperfusion and previous myocardial infarction: relation to regional myocardial function;American Journal of Physiology-Heart and Circulatory Physiology;2010-12
4. The iron-regulatory peptide hepcidin is upregulated in the ischemic and in the remote myocardium after myocardial infarction;Peptides;2010-09
5. Synergism of simvastatin with losartan prevents angiotensin II-induced cardiomyocyte apoptosis in vitro;Journal of Pharmacy and Pharmacology;2009-04-01
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