Cdk5-mediated CRMP2 phosphorylation is necessary and sufficient for peripheral neuropathic pain-Reference-Cited by-同舟云学术

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Cdk5-mediated CRMP2 phosphorylation is necessary and sufficient for peripheral neuropathic pain

Published:2019-01 Issue: Volume:5 Page:100022
ISSN:2452-073X
Container-title:Neurobiology of Pain
language:en
Short-container-title:Neurobiology of Pain

Author:

Moutal Aubin,Luo Shizhen,Largent-Milnes Tally M.,Vanderah Todd W.,Khanna Rajesh^ORCID

Funder

National Institutes of Health

Congressionally Directed Medical Research Programs

Children's Tumor Foundation

Neurofibromatosis

Publisher

Elsevier BV

Subject

Anesthesiology and Pain Medicine,Clinical Neurology,Neuroscience (miscellaneous)

Reference60 articles.

1. Phosphorylation of collapsin response mediator protein-2 by Rho-kinase. Evidence for two separate signaling pathways for growth cone collapse;Arimura;J. Biol. Chem.,2000

2. Prolyl isomerase Pin1 regulates axon guidance by stabilizing CRMP2A selectively in distal axons;Balastik;Cell Rep.,2015

3. Peripheral neuropathic pain: from mechanisms to symptoms;Baron;Clin. J. Pain,2000

4. The increased trafficking of the calcium channel subunit alpha2delta-1 to presynaptic terminals in neuropathic pain is inhibited by the alpha2delta ligand pregabalin;Bauer;J. Neurosci.,2009

5. Transcriptional and functional profiles of voltage-gated Na(+) channels in injured and non-injured DRG neurons in the SNI model of neuropathic pain;Berta;Mol. Cell Neurosci.,2008

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