Desipramine administered chronically inhibits lipopolysaccharide-stimulated production of IL-1β in the brain and plasma of rats-Reference-Cited by-同舟云学术

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Desipramine administered chronically inhibits lipopolysaccharide-stimulated production of IL-1β in the brain and plasma of rats

Published:2016-04 Issue: Volume:80 Page:26-34
ISSN:1043-4666
Container-title:Cytokine
language:en
Short-container-title:Cytokine

Author:

Obuchowicz Ewa,Prymus Agnieszka,Bielecka Anna M.,Drzyzga Łukasz,Paul-Samojedny Monika,Kot Marta,Daniel Władysława A.

Funder

Ministry of Science and Higher Education

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference74 articles.

1. Evidence for a dysregulated immune system in the etiology of psychiatric disorders;Gibney;J. Neuroimmune. Pharmacol.,2013

2. Psychoneuroimmunology meets neuropsychopharmacology: translational implications of the impact of inflammation on behavior;Haroon;Neuropsychopharmacology,2012

3. Targeting IL-1 in depression;Maes;Expert Opin. Ther. Targets,2012

4. Brain interleukin-1 mediates chronic stress-induced depression in mice via adrenocortical activation and hippocampal neurogenesis suppression;Goshen;Mol. Psychiatry,2008

5. IL-1beta is an essential mediator of the antineurogenic and anhedonic effects of stress;Koo;Proc. Natl. Acad. Sci. USA,2008

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1. Locus coeruleus injury modulates ventral midbrain neuroinflammation during DSS-induced colitis;2024-03-12

2. Locus coeruleus injury modulates ventral midbrain neuroinflammation during DSS-induced colitis;2024-02-13

3. The effects of desipramine, fluoxetine, or tianeptine on changes in bulbar BDNF levels induced by chronic social instability stress and inflammation;Pharmacological Reports;2017-06

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