IL-17A causes depression-like symptoms via NFκB and p38MAPK signaling pathways in mice: Implications for psoriasis associated depression-Reference-Cited by-同舟云学术

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IL-17A causes depression-like symptoms via NFκB and p38MAPK signaling pathways in mice: Implications for psoriasis associated depression

Published:2017-09 Issue: Volume:97 Page:14-24
ISSN:1043-4666
Container-title:Cytokine
language:en
Short-container-title:Cytokine

Author:

Nadeem Ahmed,Ahmad Sheikh F.,Al-Harbi Naif O.,Fardan Ali S.,El-Sherbeeny Ahmed M.,Ibrahim Khalid E.,Attia Sabry M.

Funder

Deanship of Scientific Research at King Saud University

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference52 articles.

1. The role of interleukin-17A in psoriatic disease;Adami;BioDrugs,2014

2. Psoriatic inflammation causes hepatic inflammation with concomitant dysregulation in hepatic metabolism via IL-17A/IL-17 receptor signaling in a murine model;Al-Harbi;Immunobiology,2017

3. Depressive and anxiety disorders in systemic lupus erythematosus patients without major neuropsychiatric manifestations;Bai;J. Immunol. Res.,2016

4. Inflammatory Th17 cells promote depression-like behavior in mice;Beurel;Biol. Psychiat.,2013

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