IL-36γ induces a transient HSV-2 resistant environment that protects against genital disease and pathogenesis-Reference-Cited by-同舟云学术

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IL-36γ induces a transient HSV-2 resistant environment that protects against genital disease and pathogenesis

Published:2018-11 Issue: Volume:111 Page:63-71
ISSN:1043-4666
Container-title:Cytokine
language:en
Short-container-title:Cytokine

Author:

Gardner Jameson K.,Herbst-Kralovetz Melissa M.

Funder

National Institutes of Health

NIAID

Valley Research Partnership

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference72 articles.

1. Correction: Global estimates of prevalent and incident herpes simplex virus type 2 infections in 2012;Looker;PLoS One,2015

2. Herpes simplex virus 2 infection increases HIV acquisition in men and women: systematic review and meta-analysis of longitudinal studies;Freeman;AIDS,2006

3. Current concepts for genital herpes simplex virus infection: diagnostics and pathogenesis of genital tract Shedding;Johnston;Clin Microbiol. Rev.,2016

4. Standard-dose and high-dose daily antiviral therapy for short episodes of genital HSV-2 reactivation: three randomised, open-label, cross-over trials;Johnston;Lancet,2012

5. Reactivation of genital herpes simplex virus type 2 infection in asymptomatic seropositive persons;Wald;New Engl. J. Med.,2000

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