Targeted inhibition of TAK1 abrogates TGFβ1 non-canonical signaling axis, NFκB/Smad7 inhibiting human endometriotic cells proliferation and inducing cell death involving autophagy

Author:

Kaushal Jyoti B.,Shukla Vinay,Sankhwar Pushplata,Jha Rajesh K.,Dwivedi Anila

Funder

Government of India Department of Biotechnology

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference69 articles.

1. Endometriosis;Bulun;N. Engl. J. Med.,2009

2. Endometriosis;Giudice;Lancet.,2004

3. Immune -Endocrine interactions in endometriosis;Cakmak;Front. Biosci. - Elit.,2009

4. Mechanistic and therapeutic implications of angiogenesis in endometriosis;Taylor;Reprod. Sci.,2009

5. A high level of TGF-B1 promotes endometriosis development via cell migration, adhesiveness, colonization, and invasiveness;Soni;Biol. Reprod.,2019

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