Cardiac dysfunction in StrepTSS: Group A streptococcus disrupts the directional cardiomyocyte-to-macrophage crosstalk that maintains macrophage quiescence

Author:

Li Zhi,Bryant Amy E.,Parimon Tanyalak,Stevens Dennis L.

Funder

United States Department of Veterans Affairs, Research and Development Service, VA Medical Center, Boise, ID

INBRE Program of the National Center for Research Resources

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference13 articles.

1. Reappearance of scarlet fever toxin A among streptococci in the Rocky Mountain West: severe group A streptococcal infections associated with a toxic shock-like syndrome;Stevens;N Eng J Med,1989

2. Clinical and microbiological characteristics of severe group A streptococcus infections and streptococcal toxic shock syndrome;Forni;Clin Infect Dis,1995

3. Case records of the Massachusetts General Hospital. Weekly Clinicopathological exercises. Case 28–2002. A 35-year-old long-term traveler with a rapidly progressive soft-tissue infection;Hoadley;N Engl J Med,2002

4. Stevens DL, Shelly M, Stiller R, Villasenor-Sierra A, Bryant AE. Acute reversible cardiomyopathy in patients with streptococcal toxic shock syndrome. In: Proceedings & Abstracts of the XVIIth Lancefield International Symposium on Streptococci and Streptococcal Diseases. Porto Heli, Greece, 2008. p. 179. (Abstract #84).

5. Proinflammatory and anti-inflammatory cytokines as mediators in the pathogenesis of septic shock;Dinarello;Chest,1997

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