To be (in a transcriptional complex) or not to be (promoting UBR5 ubiquitylation): That is an answer to how degradation controls gene expression
Author:
Funder
Max-Planck-Gesellschaft
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference10 articles.
1. UBR5 forms ligand-dependent complexes on chromatin to regulate nuclear hormone receptor stability;Tsai;Mol. Cell,2023
2. Orphan quality control shapes network dynamics and gene expression;Mark;Cell,2023
3. UBR5 Is Coamplified with MYC in Breast Tumors and Encodes an Ubiquitin Ligase That Limits MYC-Dependent Apoptosis;Qiao;Cancer Res.,2020
4. Identification of the HECT E3 ligase UBR5 as a regulator of MYC degradation using a CRISPR/Cas9 screen;Schukur;Sci. Rep.,2020
5. Gene expression and cell identity controlled by anaphase-promoting complex;Oh;Nature,2020
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1. Harnessing UBR5 for targeted protein degradation of key transcriptional regulators;Trends in Pharmacological Sciences;2023-11
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