The Methyltransferase Set7/9 (Setd7) Is Dispensable for the p53-Mediated DNA Damage Response In Vivo
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference19 articles.
1. Distinct p53 transcriptional programs dictate acute DNA-damage responses and tumor suppression;Brady;Cell,2011
2. Regulation of p53 activity through lysine methylation;Chuikov;Nature,2004
3. Functional analysis of the roles of posttranslational modifications at the p53 C terminus in regulating p53 stability and activity;Feng;Mol. Cell. Biol.,2005
4. Repression of p53 activity by Smyd2-mediated methylation;Huang;Nature,2006
5. Methylation-acetylation interplay activates p53 in response to DNA damage;Ivanov;Mol. Cell. Biol.,2007
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1. Lysine-specific methyltransferase Set7/9 in stemness, differentiation, and development;Biology Direct;2024-05-29
2. Set7/9 aggravates ischemic brain injury via enhancing glutamine metabolism in a blocking Sirt5 manner;Cell Death & Differentiation;2024-02-16
3. Cell fate regulation governed by p53: Friends or reversible foes in cancer therapy;Cancer Communications;2024-02-04
4. Tiam1 methylation by NSD2 promotes Rac1 signaling activation and colon cancer metastasis;Proceedings of the National Academy of Sciences;2023-12-19
5. SET7/9-mediated methylation affects oncogenic functions of histone demethylase JMJD2A;JCI Insight;2023-10-23
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