JNK Regulates Expression and Autocrine Signaling of TGF-β1
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference13 articles.
1. Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response
2. MEKK-1, a Component of the Stress (Stress-activated Protein Kinase/c-Jun N-terminal Kinase) Pathway, Can Selectively Activate Smad2-mediated Transcriptional Activation in Endothelial Cells
3. TGF-β signaling in tumor suppression and cancer progression
4. Smad-dependent and Smad-independent pathways in TGF-β family signalling
5. Interdependent SMAD and JNK Signaling in Transforming Growth Factor-β-mediated Transcription
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