CAG repeat expansions create splicing acceptor sites and produce aberrant repeat-containing RNAs-Reference-Cited by-同舟云学术

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CAG repeat expansions create splicing acceptor sites and produce aberrant repeat-containing RNAs

Published:2024-02 Issue:4 Volume:84 Page:702-714.e10
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Anderson Rachel,Das Michael R.,Chang Yeonji,Farenhem Kelsey,Schmitz Cameron O.,Jain Ankur^ORCID

Publisher

Elsevier BV

Reference84 articles.

1. Molecular mechanisms underlying nucleotide repeat expansion disorders;Malik;Nat. Rev. Mol. Cell Biol.,2021

2. Repeat expansion diseases;Paulson;Handb. Clin. Neurol.,2018

3. Mutant CAG repeats of Huntingtin transcript fold into hairpins, form nuclear foci and are targets for RNA interference;de Mezer;Nucleic Acids Res.,2011

4. Cellular toxicity of expanded RNA repeats: focus on RNA foci;Wojciechowska;Hum. Mol. Genet.,2011

5. RNA phase transitions in repeat expansion disorders;Jain;Nature,2017

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. CircHTT(2,3,4,5,6) — co-evolving with the HTT CAG-repeat tract — modulates Huntington's disease phenotypes;Molecular Therapy - Nucleic Acids;2024-09

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