Disease-Causing Mutations in SF3B1 Alter Splicing by Disrupting Interaction with SUGP1-Reference-Cited by-同舟云学术

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Disease-Causing Mutations in SF3B1 Alter Splicing by Disrupting Interaction with SUGP1

Published:2019-10 Issue:1 Volume:76 Page:82-95.e7
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Zhang Jian,Ali Abdullah M.,Lieu Yen K.,Liu Zhaoqi,Gao Jianchao,Rabadan Raul,Raza Azra,Mukherjee Siddhartha,Manley James L.

Funder

NIH

EvansMDS

Partnership for Cures

NCI

Herbert Irving Comprehensive Cancer Center

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference73 articles.

1. Evolving new protein-protein interaction specificity through promiscuous intermediates;Aakre;Cell,2015

2. Cancer-associated SF3B1 mutations affect alternative splicing by promoting alternative branchpoint usage;Alsafadi;Nat. Commun.,2016

3. Stabilization of protein-protein interactions in drug discovery;Andrei;Expert Opin. Drug Discov.,2017

4. G-patch: a new conserved domain in eukaryotic RNA-processing proteins and type D retroviral polyproteins;Aravind;Trends Biochem. Sci.,1999

5. Chronic immune response dysregulation in MDS pathogenesis;Barreyro;Blood,2018

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3. The E592K variant of SF3B1 creates unique RNA missplicing and associates with high-risk MDS without ring sideroblasts;Blood Advances;2024-07-26

4. (G)Patching up mis-splicing in cancer;Trends in Biochemical Sciences;2024-07

5. Exploring the impact of lipid droplets on the evolution and progress of hepatocarcinoma;Frontiers in Cell and Developmental Biology;2024-05-16

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